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Journal of Alzheimer's Disease , 2018

[10] Clinical studies that are shared in this article show that this initial disruption to the skin barrier in the follicular canal leads to many downstream symptoms such as defective skin cell shedding, atrophied sebaceous glands, trans epidermal water loss, hair shaft abnormalities, inflammation, and post inflammatory hyper-pigmentation

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Intriguingly, antioxidant defense mechanisms in the CNS diverge substantially from those in peripheral tissues such as erythrocytes, where the glutathioneNADPH system is well characterized and robust